Differential 3' splice site recognition of SMN1 and SMN2 transcripts by U2AF and U2 snRNP

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Differential 3' splice site recognition of SMN1 and SMN2 transcripts by U2AF and U2 snRNP.

Spinal Muscular atrophy is a prevalent genetic disease caused by mutation of the SMN1 gene, which encodes the SMN protein involved in assembly of small nuclear ribonucleoprotein (snRNP) complexes. A paralog of the gene, SMN2, cannot provide adequate levels of functional SMN because exon 7 is skipped in a significant fraction of the mature transcripts. A C to T transition located at position 6 o...

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U4/U5/U6 snRNP recognizes the 5' splice site in the absence of U2 snRNP.

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Intron removal requires proofreading of U2AF/3' splice site recognition by DEK.

Discrimination between splice sites and similar, nonsplice sequences is essential for correct intron removal and messenger RNA formation in eukaryotes. The 65- and 35-kD subunits of the splicing factor U2AF, U2AF65 and U2AF35, recognize, respectively, the pyrimidine-rich tract and the conserved terminal AG present at metazoan 3' splice sites. We report that DEK, a chromatin- and RNA-associated ...

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ژورنال

عنوان ژورنال: RNA

سال: 2009

ISSN: 1355-8382

DOI: 10.1261/rna.1273209